Evidence mapPaperPMID 42467086Full record

ArticleDiabetologia2026

Dapagliflozin does not impair LDL catabolism and induces a shift towards a closer to normal VLDL

Bruno Vergès, Alexia Rouland, Benjamin Bouillet, Jean-Paul Pais de Barros, Coralie Fourmont, Perrine Buffier, Tony Jourdan, Victoria Vergas, Laurence Duvillard

Registry-linked trialAbstract read
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In one paragraph

Article in Diabetologia, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It reports registered trial NCT03269058. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT03269058 phase4completed

Effects of Dapagliflozin on Lipoprotein Kinetics in Patients With Type 2 Diabetes

Ran2017Enrolled28Registered outcomes9Posted comparisons0ConditionsOral Antidiabetics, Type-2 DiabetesArmsdapagliflozin, Placebos
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3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Bruno VergèsUniversité de Bourgogne Europe, Inserm UMR1231 Center for Translational and Molecular Medicine (CTM), Team Pathophysiology of Dyslipidemia (PADYS), Dijon, France. bruno.verges@chu-dijon.fr.
Alexia RoulandUniversité de Bourgogne Europe, Inserm UMR1231 Center for Translational and Molecular Medicine (CTM), Team Pathophysiology of Dyslipidemia (PADYS), Dijon, France.
Benjamin BouilletUniversité de Bourgogne Europe, Inserm UMR1231 Center for Translational and Molecular Medicine (CTM), Team Pathophysiology of Dyslipidemia (PADYS), Dijon, France.
Jean-Paul Pais de BarrosUniversité Bourgogne Europe, Inserm, UMS 058 BioSanD, DiviOmics, Dijon, France.
Coralie FourmontUniversité de Bourgogne Europe, CHU Dijon Bourgogne, Department of Endocrinology/Diabetology, Dijon, France.
Perrine BuffierUniversité de Bourgogne Europe, CHU Dijon Bourgogne, Department of Endocrinology/Diabetology, Dijon, France.
Tony JourdanUniversité de Bourgogne Europe, Inserm UMR1231 Center for Translational and Molecular Medicine (CTM), Team Pathophysiology of Dyslipidemia (PADYS), Dijon, France.
Victoria VergasUniversité Bourgogne Europe, Inserm, UMS 058 BioSanD, DiviOmics, Dijon, France.
Laurence DuvillardUniversité de Bourgogne Europe, Inserm UMR1231 Center for Translational and Molecular Medicine (CTM), Team Pathophysiology of Dyslipidemia (PADYS), Dijon, France.

Funding

AstraZeneca France grantsfrom AstraZenecaFrench National Research Agency (ANR) Program "Investissements d'Avenir" with refeRégion Bourgogne-Franche Comté Fonds Européens de Développement Régional
6 · The paper itself

Abstract

aims/hypothesisSGLT2 inhibitors reduce atherosclerosis, but their effect on lipid metabolism remains unclear. Animal studies have suggested that SGLT2 inhibitors may decrease LDL catabolism by reducing the hepatic expression of LDL receptors. However, the effect of SGLT2 inhibitors on LDL metabolism, and more widely on all ApoB-containing lipoproteins, remain unknown in humans. This prompted us to study the effect of the SGLT2 inhibitor dapagliflozin on the metabolism of ApoB100-containing lipoproteins in individuals with type 2 diabetes.

methodsWe performed a parallel double-blinded in vivo kinetic study using stable isotopes (tri-deuterated [

resultsCompared with placebo-treated individuals (n=7), those who received dapagliflozin (n=17) showed a significant reduction of body weight (p<0.001) and HbA CONCLUSIONS/

interpretationDapagliflozin does not impair the catabolism of LDL in individuals with type 2 diabetes. It reduces direct catabolism of VLDL

trial registrationClinicalTrials.gov NCT03269058

fundingThe study was funded via grants and support from AstraZeneca, the French National Research Agency (ANR) under the programme 'Investissements d'Avenir', the University of Burgundy-Franche-Comté, the National Institute of Health and Medical Research (Inserm), the Region Burgundy - Franche Comté, and the Fonds Européens de Développement Régional.

Indexed as

DapagliflozinKineticLDLLipoproteinSGLT2 inhibitorVLDLVLDL1

Identifiers

What Socratic holds

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Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.