Evidence mapPaperPMID 42468113Full record

ArticleEBioMedicine2026

GnRH-receptor antagonism as a targeted approach to reproductive dysfunction in polycystic ovary syndrome.

Ludovica Cotellessa, Hélène Maitre, Frank Giton, Silvia Bongiovanni, Pascal Pigny, Geoffroy Robin, Sophie Catteau-Jonard, Paolo Giacobini

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Article in EBioMedicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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4 · The record

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5 · Who and what money

Authors and funding

8 authors.

Ludovica CotellessaInserm, CHU Lille, Lille Neuroscience & Cognition, UMR-S 1172, Univ. Lille, Lille, France.
Hélène MaitreInserm, CHU Lille, Lille Neuroscience & Cognition, UMR-S 1172, Univ. Lille, Lille, France.
Frank GitonAP-HP, Pôle Biologie-Pathologie Henri Mondor, 94010, Créteil, France; Inserm IMRB U955, 94010, Créteil, France.
Silvia BongiovanniInserm, CHU Lille, Lille Neuroscience & Cognition, UMR-S 1172, Univ. Lille, Lille, France.
Pascal PignyCHU Lille, Service de Biochimie et Hormonologie, Centre de Biologie Pathologie, Lille, France.
Geoffroy RobinCHU Lille, Service de Gynécologie Médicale, Hôpital Jeanne de Flandre, Lille, France.
Sophie Catteau-JonardInserm, CHU Lille, Lille Neuroscience & Cognition, UMR-S 1172, Univ. Lille, Lille, France; CHU Lille, Service de Gynécologie Médicale, Hôpital Jeanne de Flandre, Lille, France. Electronic address: Sophie.CATTEAU@chu-lille.fr.
Paolo GiacobiniInserm, CHU Lille, Lille Neuroscience & Cognition, UMR-S 1172, Univ. Lille, Lille, France. Electronic address: paolo.giacobini@inserm.fr.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundPolycystic ovary syndrome (PCOS), recently renamed polyendocrine metabolic ovarian syndrome (PMOS), is characterised by neuroendocrine dysfunction with accelerated gonadotrophin-releasing hormone (GnRH)/luteinising hormone (LH) pulsatility driving hyperandrogenism and anovulatory infertility.

methodsWe used the prenatal anti-Müllerian hormone (AMH)-exposed PMOS-like mouse model (PAMH) and a phase I clinical trial in women with PMOS without obesity. PAMH and control mice received acute or intermittent low-dose Ganirelix, and oestrous cyclicity, ovulation, gonadotrophins, and steroids were assessed. In women, two subtherapeutic Ganirelix doses (0.025 mg, n = 8; 0.0625 mg, n = 10) were administered once in early follicular phase, with 10-min blood sampling over 8 h to quantify LH pulsatility and reproductive hormones.

findingsIn PMOS-like mice, a single Ganirelix injection normalised exaggerated LH pulsatility, and six-week intermittent treatment restored oestrous cyclicity, ovulation, and testosterone levels without affecting controls. In women with PMOS both Ganirelix doses reduced LH pulse frequency, basal, mean and total LH, and decreased the LH/FSH ratio. D4-androstenedione fell by 25-30% at both doses, AMH declined modestly at 0.0625 mg, while oestradiol remained unchanged.

interpretationLow-dose GnRH-receptor antagonism with Ganirelix can recalibrate, rather than suppress, GnRH/LH signalling, attenuating hyperandrogenism and, in mice, restoring ovulatory function. These data identify partial GnRHR blockade as a promising neuroendocrine-centred strategy in PMOS and provide a rationale for phase II trials evaluating repeated low-dose regimens, ovulatory restoration, and fertility outcomes.

fundingThis work was supported by the European Research Council (ERC) Horizon-ERC-POC grant (ERC-2022-POC2, n° 101111874) and the French National Research Agency (ANR-24-CHBS-0002, France 2030).

Indexed as

Polycystic Ovary SyndromeReceptors, LHRHReproductionAdultAnimalsDisease Models, AnimalEstrous CycleFemaleGonadotropin-Releasing HormoneHumansHypothalamic-Pituitary-Gonadal AxisLuteinizing HormoneMiceganirelixGonadotropin-Releasing HormoneLuteinizing HormoneReceptors, LHRHFertilityGanirelixGnRHHyperandrogenismLHPolycystic ovary syndrome (PCOS)Polyendocrine metabolic ovarian syndrome (PMOS)

Identifiers

PMID42468113
PMCPMC13400290

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.