Evidence map›Paper›PMID 42494597›Full record

ReviewFrontiers in oncology2026

Translating physical stress into biological signals: precision electro-oncology based on conformational changes of electro-sensitive receptors and parameterized immune remodeling.

Wei Shi, Mengya Zhao, Xiaofeng Ma, Li Dong, Yulong Sun

Abstract readReview
In one paragraph

Review in Frontiers in oncology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Wei Shi *Department of Urology, The Second Hospital of Lanzhou University, and Gansu Province Clinical Research Center for Urinary System Disease, Lanzhou, China.
Mengya Zhao *School of Life Science and Technology, Key Laboratory for Space Biosciences & Biotechnology, Institute of Special Environmental Biophysics, Research Center of Special Environmental Biomechanics and Medical Engineering, Engineering Research Center of Chinese Ministry of Education for Biological Diagnosis, Treatment and Protection Technology and Equipment, Northwestern Polytechnical University, Xi'an, Shaanxi, China.
Xiaofeng MaSchool of Life Science and Technology, Key Laboratory for Space Biosciences & Biotechnology, Institute of Special Environmental Biophysics, Research Center of Special Environmental Biomechanics and Medical Engineering, Engineering Research Center of Chinese Ministry of Education for Biological Diagnosis, Treatment and Protection Technology and Equipment, Northwestern Polytechnical University, Xi'an, Shaanxi, China.
Li DongNHC Key Laboratory of Diagnosis and Therapy of Gastrointestinal Tumor, Gansu Provincial Hospital, Lanzhou, China.
Yulong SunSchool of Life Science and Technology, Key Laboratory for Space Biosciences & Biotechnology, Institute of Special Environmental Biophysics, Research Center of Special Environmental Biomechanics and Medical Engineering, Engineering Research Center of Chinese Ministry of Education for Biological Diagnosis, Treatment and Protection Technology and Equipment, Northwestern Polytechnical University, Xi'an, Shaanxi, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Tumor Treating Fields (TTFields) and pulsed electric fields (PEF) have emerged as a fourth modality in oncological treatment, succeeding surgery, radiotherapy, and pharmacotherapy. However, the fundamental mechanisms through which physical electrical stress is precisely transduced into biochemical signals remain to be fully elucidated. By integrating molecular dynamics simulations, multi-omics profiling, and the latest clinical trial data, this review systematically explores how electric fields, functioning as physical signals, initiate intracellular transduction by inducing conformational kinetic changes in G protein-coupled receptors (GPCRs), such as NPFFR2. Furthermore, we discuss how the modulation of waveform parameters enables the precise programming of apoptosis and pyroptosis patterns, thereby inducing immunogenic cell death (ICD) and activating the cGAS-STING pathway. Additionally, proteomics-based insights are employed to reveal the mechanisms of electric-field-induced compensatory resistance, and the pivotal role of electric fields in remodeling the tumor immune microenvironment and reversing resistance to immune checkpoint inhibitors is discussed. This review aims to provide refined theoretical guidance for the clinical translation of the emerging field of the "electro-immunome".

Indexed as

bioelectric signalselectro-sensitive receptorsphysical signaltumor immune microenvironmenttumor-treating fields

Identifiers

PMID42494597
PMCPMC13391412

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.