ReviewMedicine2026
A bibliometric analysis of the pathophysiology of sepsis-associated acute kidney injury from 2015 to 2024.
Review in Medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
Abstract
backgroundThe pathophysiology of sepsis-associated acute kidney injury (SA-AKI) urgently needs to be further explored to translate these findings into potential early diagnostic methods and effective therapeutic strategies. This study aims to analyze the current landscape and development trend related to the pathophysiology of SA-AKI.
methodsNetwork visualization and bibliometric analysis were conducted using the "Citation Report" function in Web of Science, CiteSpace, VOSviewer, the bibliometric package in R and the online platform (https://bibliometric.com), based on the publications from 2015 to 2024 in this field from the Web of Science Core Collection database.
resultsOver the past decade, the number of publications and citations per year has steadily increased in this research area. China was far ahead in the number of publications (n = 690, 71.06%), followed by the United States (n = 123, 12.67%). The United States had the most extensive national collaboration. Seven of the top 10 most prolific institutions were situated in China. The 3 most productive authors were Bellomo (n = 16), Kellum (n = 14) and Gomez (n = 13). Bagshaw was the most cited author. International Immunopharmacology, Shock and Inflammation published the most papers. The review published in Kidney International by Peerapornratana et al in 2019 ranked first both in citation and co-citation frequency. The mechanisms of SA-AKI mainly included inflammation, microcirculation dysfunction, metabolic reprogramming and mitochondrial dysfunction. Metabolic reprogramming and mitochondrial dysfunction are still research hotspots at present. Pyroptosis and ferroptosis have emerged as research frontiers.
conclusionIn summary, this research comprehensively analyzed the research status and development trend in the field of the pathophysiology of SA-AKI by bibliometric analysis. The findings can offer valuable information for scholars seeking academic cooperation and identifying future research directions.
Indexed as
Identifiers
What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.