ReviewFrontiers in cell and developmental biology2026
The molecular mechanisms of gas signaling molecules in bronchopulmonary dysplasia related lung injury.
Review in Frontiers in cell and developmental biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Bronchopulmonary dysplasia (BPD) is the most prevalent chronic respiratory disease in preterm infants. Its pathogenesis is linked to multiple factors including lung immaturity secondary to preterm birth, hyperoxia exposure, mechanical ventilation-induced injury, and inflammatory responses. Characterized by alveolar simplification and abnormal pulmonary vascular development as the core pathological features, BPD exerts a profound negative impact on the long-term prognosis of affected infants. As endogenous small-molecule substances, gas signaling molecules play a pivotal role in regulating BPD related lung injury by virtue of their extensive biological properties. Among these molecules, nitric oxide (NO), carbon monoxide (CO), and hydrogen sulfide (H
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