ReviewFrontiers in immunology2026
Metabolic reprogramming orchestrates microglial fate in cerebral ischemia-reperfusion injury: a spatiotemporal immunometabolic perspective.
Review in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Abstract
Cerebral ischemia-reperfusion injury (CIRI) represents a critical pathological cascade that paradoxically exacerbates neurological damage following revascularization therapy for acute ischemic stroke (AIS). The pathogenesis of CIRI is intricately linked to dysregulated neuroinflammation, with microglia-the resident innate immune cells of the central nervous system-serving as central orchestrators of this response. Emerging evidence indicates that microglia undergo profound metabolic reprogramming encompassing glucose metabolism, the tricarboxylic acid (TCA) cycle, fatty acid metabolism, and NAD
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