Evidence mapPaperPMID 42564337Full record

ArticleHepatology forum2026

Empagliflozin versus dapagliflozin in patients with liver cirrhosis: A comparative real-world study on hepatic decompensation.

Hatem Ahmed, Imad Alabdul Razzak, Eyad Abdulrazzak, Sameh Gomaa, Joelle Lauchner

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Article in Hepatology forum, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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4 · The record

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5 · Who and what money

Authors and funding

5 authors.

Hatem AhmedDepartment of Internal Medicine Residency, Phoenixville Hospital, Pennsylvania, USA.
Imad Alabdul RazzakDepartment of Internal Medicine Residency, Phoenixville Hospital, Pennsylvania, USA.
Eyad AbdulrazzakBeth Israel Deaconess Medical Center- Boston, Massachusetts, USA.
Sameh GomaaDepartment of Internal Medicine Residency, Phoenixville Hospital, Pennsylvania, USA.
Joelle LauchnerTower Health Medical Group Internal Medicine, Douglassville, Pennsylvania, USA.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background and Aim: Sodium-glucose cotransporter-2 inhibitors may improve outcomes in liver cirrhosis, but comparative evidence between individual agents is limited. We compared real-world outcomes of empagliflozin versus dapagliflozin in adults with liver cirrhosis. Materials and Methods: We conducted a multicenter retrospective cohort study using the TriNetX US Collaborative Network (69 healthcare organizations). Adults with cirrhosis who were newly prescribed empagliflozin or dapagliflozin after the diagnosis of cirrhosis between March 1, 2013 and January 1, 2025, were included. Propensity score matching (1:1) was used to balance baseline characteristics. Outcomes were assessed from day 1 after the index prescription through 5 years. Primary outcomes were all-cause mortality and all-cause hospitalization. Secondary outcomes included hepatic decompensation events. Tertiary outcomes included prognostic hepatic and renal biomarkers. Safety outcomes included the incidence of acute kidney injury, urinary tract infection, and diabetic ketoacidosis. Results: Before matching, 17,700 empagliflozin users and 8,619 dapagliflozin users were identified; 7,852 patients remained in each cohort after matching. Five-year mortality was similar between groups (12.7% vs. 12.6%; odds ratio [OR]: 1.012, 95% confidence interval [CI]: 0.921-1.112; p=0.8075), as was the risk of hospitalization (14.7% vs. 13.4%; OR: 1.105, 95% CI: 0.94-1.30; p=0.216). Empagliflozin was associated with lower rates of hepatic encephalopathy (4.0% vs. 4.7%; OR: 0.84; p=0.0295), hepatorenal syndrome (1.0% vs. 1.6%; OR: 0.614; p=0.0007), and paracentesis (2.9% vs. 3.7%; OR: 0.785; p=0.0083). Albumin levels were higher and bilirubin levels were lower with empagliflozin (p<0.01 for both). Safety outcomes were similar between groups. Conclusion: In matched adults with cirrhosis, empagliflozin and dapagliflozin demonstrated comparable five-year mortality and hospitalization rates. However, empagliflozin was associated with fewer selected decompensation events without an increase in adverse events.

Indexed as

Cirrhosisdapagliflozinempagliflozin

Identifiers

PMID42564337
PMCPMC13442726

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.