Evidence mapPaperPMID 42570045Full record

ArticleEndocrine2026

Functional causes of low testosterone predominate in contemporary Australian endocrine referral practice: a two centre experience across 5 years.

Aongus O'Brolchain, Kacie McAndrew, William Newman, Dylan Young, Dian Van Zyl, Randunu Dissanyake, Elahe Ebrahimi, Katherine Griffin

Abstract readMulticenter Study
In one paragraph

Article in Endocrine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

8 authors.

Aongus O'BrolchainGriffith University, Southport, QLD, Australia. a.obrolchain@griffith.edu.au.ORCID https://orcid.org/0009-0006-8753-6386
Kacie McAndrewGold Coast Hospital and Health Service, Southport, QLD, Australia.
William NewmanGold Coast Hospital and Health Service, Southport, QLD, Australia.
Dylan YoungGold Coast Hospital and Health Service, Southport, QLD, Australia.
Dian Van ZylGold Coast Hospital and Health Service, Southport, QLD, Australia.
Randunu DissanyakeGold Coast Hospital and Health Service, Southport, QLD, Australia.
Elahe EbrahimiGold Coast Hospital and Health Service, Southport, QLD, Australia.
Katherine GriffinGold Coast Hospital and Health Service, Southport, QLD, Australia.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundThe epidemiology and outpatient management of low testosterone in Australian tertiary care remain poorly characterised. Rising testosterone prescription rates and evolving guidelines underscore the need to delineate contemporary referral patterns, diagnostic adherence, and therapeutic practice.

methodsA retrospective review was undertaken of all new male patients assessed for low testosterone levels at Gold Coast University and Robina Hospitals between 2020 and 2024. Demographic, biochemical, and management data were extracted from the integrated electronic medical record. Continuous variables were analysed using the Kruskal-Wallis test, categorical variables using χ² or Fisher's exact test, and binary logistic regression identified predictors of testosterone replacement therapy (TRT) initiation and pituitary imaging.

resultsAmong 294 consecutive referrals, functional low testosterone (43%) exceeded all pathological diagnostic categories combined individually. Obesity affected 65% of patients and obstructive sleep apnoea 28%, the latter highest in functional low testosterone (48%; p < 0.001). Two-thirds of referrals included two early-morning testosterone samples, pituitary imaging was performed in one-quarter of patients, with clinically actionable abnormalities identified in 4% of imaged men. TRT was prescribed in 25% of men, less often in functional than pathological forms (23% vs. 45%; OR 0.37, 95% CI 0.22-0.62; p < 0.001). Most patients were discharged after a median of three visits and six months' follow-up. Functional low testosterone was associated with a high burden of cardiometabolic and psychological comorbidity. Across the entire cohort, 31% reported previous androgen exposure.

conclusionsMost men referred with low testosterone did not have structural hypothalamic-pituitary-testicular axis disease, supporting a clinical approach prioritising identification of metabolic and reversible contributors before testosterone therapy. Diagnostic completeness and adherence to guideline-recommended imaging vary, but testosterone prescribing generally aligns with current Endocrine Society of Australia (ESA) and Pharmaceutical Benefits Scheme (PBS) restrictions. These findings support the establishment of a dedicated andrology service integrating metabolic, reproductive, and psychological care to optimise assessment and outcomes in men with androgen deficiency.

Indexed as

HypogonadismTestosteroneAdultAgedAustraliaHormone Replacement TherapyHumansMaleMiddle AgedObesityPituitary GlandReferral and ConsultationRetrospective StudiesTestosteroneAndrologyHypogonadismTestosterone

Identifiers

PMID42570045
PMCPMC13452758

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.