Evidence map›Paper›PMID 42572615›Full record

ArticleiScience2026

IL-10-producing innate lymphoid cells restrain γδT17-driven psoriatic inflammation.

Min Geun Jo, Min Yeong Choi, Keun Young Min, Jeong Won Park, Juhyun Shin, Ji-Ae Lee, Sunsook Hwang, Geunwoong Noh, Young Mi Kim, Wahn Soo Choi and 1 more

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in iScience, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Min Geun JoDepartment of Immunology, School of Medicine, Konkuk University, Chungju 27478, Korea.
Min Yeong ChoiDepartment of Immunology, School of Medicine, Konkuk University, Chungju 27478, Korea.
Keun Young MinDepartment of Microbiology and Immunology, Weill Cornell Medicine, New York, NY 10021, USA.
Jeong Won ParkDepartment of Biomedical Sciences, College of Natural Science and Department of Health Sciences, The Graduate School of Dong-A University, Busan 49315, Korea.
Juhyun ShinDepartment of Biomedical Sciences, College of Natural Science and Department of Health Sciences, The Graduate School of Dong-A University, Busan 49315, Korea.
Ji-Ae LeeDepartment of Biomedical Sciences, College of Natural Science and Department of Health Sciences, The Graduate School of Dong-A University, Busan 49315, Korea.
Sunsook HwangDepartment of Immunology, School of Medicine, Konkuk University, Chungju 27478, Korea.
Geunwoong NohDepartment of Allergy, Allergy and Clinical Immunology Center, Cheju Halla General Hospital, Jeju 63127, Korea.
Young Mi KimDepartment of Preventive Pharmacy, College of Pharmacy, Duksung Women's University, Seoul 01369, Korea.
Wahn Soo ChoiDepartment of Immunology, School of Medicine, Konkuk University, Chungju 27478, Korea.
Hyuk Soon KimDepartment of Biomedical Sciences, College of Natural Science and Department of Health Sciences, The Graduate School of Dong-A University, Busan 49315, Korea.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Psoriasis is a chronic inflammatory dermatosis driven by the interleukin (IL)-23-IL-17 axis, with γδ T cells as key IL-17A producers in lesional skin. IL-10 counter-regulates this pathway, and innate lymphoid cells (ILCs) are emerging tissue-resident immune regulators. Here we show that imiquimod (IMQ)-induced psoriatic inflammation elicits a programmed death-ligand 1 (PD-L1)

Indexed as

CCR2IL-10psoriasisregulatory ILCsγδ T cells

Identifiers

PMID42572615
PMCPMC13453000

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.