Evidence mapPaperPMID 42577639Full record

ArticleExperimental and therapeutic medicine2026

Crocin I delays cellular senescence potentially via modulating SIRT1/PKM2 expression to improve lactate homeostasis

Yicheng Fu, Zhiwei Ding, Yunchuang Chang, Chunxia Wu, Sanying Wang, Genxiang Mao, Huili Su

Abstract read
In one paragraph

Article in Experimental and therapeutic medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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4 · The record

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5 · Who and what money

Authors and funding

7 authors.

Yicheng FuCollege of Life Science, Zhejiang Chinese Medical University, Hangzhou, Zhejiang 310053, P.R. China.
Zhiwei DingZhejiang Key Laboratory of Geriatrics, Geriatrics Institute of Zhejiang Province, Zhejiang Hospital, Hangzhou, Zhejiang 310030, P.R. China.
Yunchuang ChangZhejiang Key Laboratory of Geriatrics, Geriatrics Institute of Zhejiang Province, Zhejiang Hospital, Hangzhou, Zhejiang 310030, P.R. China.
Chunxia WuCollege of Life Science, Zhejiang Chinese Medical University, Hangzhou, Zhejiang 310053, P.R. China.
Sanying WangZhejiang Key Laboratory of Geriatrics, Geriatrics Institute of Zhejiang Province, Zhejiang Hospital, Hangzhou, Zhejiang 310030, P.R. China.
Genxiang MaoCollege of Life Science, Zhejiang Chinese Medical University, Hangzhou, Zhejiang 310053, P.R. China.
Huili SuDepartment of Pharmacy, Zhejiang Hospital, Hangzhou, Zhejiang 310030, P.R. China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Crocin I has a protective effect against D-galactose-induced aging in rat models; however, its specific mechanism on delaying cellular senescence remains unclear. The aim of the present study was to investigate the interventional effect and related molecular mechanism of Crocin Ⅰ on replicative senescence and premature senescence of human embryonic lung diploid fibroblasts (WI-38) cells. Replicative senescence model was established through long-term culture and passaging to >55 PD. The premature senescence model was established using WI-38 cells treated with 2,2'-azobis-2-methy-propanimidamide dihydrochloride. The cell viability was detected by using a cell counting kit-8 assay, and the effects of Crocin Ⅰ on senescent phenotype of WI-38 cells were detected by senescence-associated β-galactosidase (SA-β-Gal) staining. In addition, the intracellular reactive oxygen species (ROS) level was detected by flow cytometry, and gene expression levels were detected by quantitative PCR. RNA sequencing analysis was performed to explore the alteration genes in senescent cells and the Crocin Ⅰ intervention group. The proteins PKM2, SIRT1, p53 and p21 in WI-38 cells were detected by western blotting, whereas cellular lactate level was detected by L-lactic acid content detection kit and pyruvate kinase (PK) enzyme activity was measured using PK activity assay kit. Crocin Ⅰ promoted cell proliferation within the range of 10-40 µM. In addition, Crocin Ⅰ decreased the SA-β-Gal staining positive rate on both replicative and premature senescent WI-38 cells. Furthermore, the intracellular ROS level was decreased by Crocin Ⅰ intervention. The differentially expressed genes were mainly enriched in phosphorylation, oxidation-reduction process, glucose metabolic process, cellular senescence pathway, glycolysis, hippo signaling pathway and pyruvate metabolism. RNA sequencing analysis and molecular docking revealed that PKM2 was one of the potential targets in Crocin Ⅰ against senescence activity. Furthermore, Crocin Ⅰ could enhance the expression of SIRT1 following with decreasing the level of PKM2, p21 and p53. Meanwhile, Crocin Ⅰ significantly decreased the cellular lactate level in WI-38. In conclusion, Crocin Ⅰ alleviated the replicative and premature senescence of WI-38 cells partially through regulating SIRT1 and PKM2 expression. Furthermore, this function may involve in glycolysis pathway.

Indexed as

cell senescencecrocin ⅠglycolysisPKM2SIRT1

Identifiers

PMID42577639
PMCPMC13454989

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.