Evidence mapPaperPMID 42583887Full record

ReviewEuropean journal of clinical investigation2026

Differential Effects of Tafamidis on Cardiac Remodelling in Transthyretin Amyloid Cardiomyopathy: Evidence From Contemporary Imaging Data.

Tameem Quader, Krisha Patel, James Sindone, Shaun Khanna, Aditya Bhat, Henry Chen, Gary Gan, Nitesh Nerlekar, Suzanne Eshoo, Nicole Bart and 2 more

Abstract readReview
In one paragraph

Review in European journal of clinical investigation, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Tameem QuaderDepartment of Cardiology, Blacktown Hospital, New South Wales, Australia.ORCID https://orcid.org/0009-0009-4983-2055
Krisha PatelSchool of Medicine, Western Sydney University, New South Wales, Australia.ORCID https://orcid.org/0009-0002-4571-4245
James SindoneDepartment of Cardiology, Royal Prince Alfred Hospital, New South Wales, Australia.
Shaun KhannaCardiovascular Program, The George Institute for Global Health, New South Wales, Australia.ORCID https://orcid.org/0000-0002-6741-1096
Aditya BhatDepartment of Cardiology, Blacktown Hospital, New South Wales, Australia.
Henry ChenDepartment of Cardiology, Blacktown Hospital, New South Wales, Australia.
Gary GanDepartment of Cardiology, Blacktown Hospital, New South Wales, Australia.ORCID https://orcid.org/0000-0001-6430-2743
Nitesh NerlekarVictorian Heart Institute, Victorian Heart Hospital, Victoria, Australia.ORCID https://orcid.org/0000-0002-3437-8648
Suzanne EshooDepartment of Cardiology, Blacktown Hospital, New South Wales, Australia.ORCID https://orcid.org/0000-0002-0018-7309
Nicole BartDepartment of Cardiology, St Vincents Hospital, New South Wales, Australia.
Liza ThomasDepartment of Cardiology, Westmead Hospital, New South Wales, Australia.ORCID https://orcid.org/0000-0001-6045-7780
Andrew SindoneDepartment of Cardiology, Concord Repatriation General Hospital, New South Wales, Australia.ORCID https://orcid.org/0000-0003-0075-805X

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundTafamidis is the only approved disease-modifying therapy for transthyretin amyloid cardiomyopathy (ATTR-CM) in Australia. Although its clinical efficacy is established, its effects on cardiac remodelling assessed by contemporary multimodality imaging remain incompletely defined.

methodsMEDLINE, Embase and Cochrane databases were systematically searched from 2000 to 2025. Observational studies evaluating tafamidis-treated versus untreated/control patients that reported serial TTE, CMR or bone-tracer scintigraphy measurements were eligible. Change-from-baseline values were used to derive measures. Outcomes were pooled as standardised mean differences (SMDs) with 95% confidence intervals using random-effects modelling. A two-sided p < 0.05 was considered statistically significant.

resultsOver a mean follow-up of 16 months, tafamidis was associated with significant improvements in left atrial reservoir strain (LASr) (SMD +0.58, 95% CI +0.27 to +0.89; p < 0.001; I

conclusionsTafamidis demonstrates selective remodelling effects in ATTR-CM, with the most benefits noted in left atrial function and RV systolic performance, rather than LV structural or functional parameters. Imaging parameters such as LASr, LAVi, and RVEF may serve as sensitive endpoints of therapeutic response in future clinical trials, offering mechanistic insights beyond conventional LV metrics. STUDY REGISTRATION: PROSPERO CRD420251145891.

Indexed as

Amyloid Neuropathies, FamilialBenzoxazolesCardiomyopathiesVentricular RemodelingEchocardiographyHumansMagnetic Resonance ImagingStroke VolumeBenzoxazolestafamidisbone‐tracer cardiac scintigraphycardiac magnetic resonanceechocardiographyTafamidistransthyretin amyloid cardiomyopathy

Identifiers

PMID42583887
PMCPMC13463392

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.