Evidence map›Paper›PMID 42592945›Full record

ArticleFEBS open bio2026

GelMA-based 3D spheroids recapitulate transcriptomic and functional hallmarks of myeloid sarcoma.

Nicolas Germain, Elise Buret, Le Hoang Thanh Nguyen, Marion Cocheteux, Nicolas Touya, Mélanie Dhayer, Romain Dubois, Frank Lafont, Sebastien Janel, Patrice Maboudou and 2 more

Abstract read
In one paragraph

Article in FEBS open bio, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Nicolas GermainUniversity of Lille, CNRS, Inserm, CHU Lille, Institut de Recherche Contre le Cancer de Lille, UMR9020 - UMR-S 1277 - Canther Cancer Heterogeneity, Plasticity and Therapy Resistance, France.ORCID https://orcid.org/0000-0003-0867-3245
Elise BuretUniversity of Lille, CNRS, Inserm, CHU Lille, Institut de Recherche Contre le Cancer de Lille, UMR9020 - UMR-S 1277 - Canther Cancer Heterogeneity, Plasticity and Therapy Resistance, France.
Le Hoang Thanh NguyenUniversity of Lille, CNRS, Inserm, CHU Lille, Institut de Recherche Contre le Cancer de Lille, UMR9020 - UMR-S 1277 - Canther Cancer Heterogeneity, Plasticity and Therapy Resistance, France.
Marion CocheteuxUniversity of Lille, CNRS, Inserm, CHU Lille, Institut de Recherche Contre le Cancer de Lille, UMR9020 - UMR-S 1277 - Canther Cancer Heterogeneity, Plasticity and Therapy Resistance, France.
Nicolas TouyaUniversity of Lille, CNRS, Inserm, CHU Lille, Institut de Recherche Contre le Cancer de Lille, UMR9020 - UMR-S 1277 - Canther Cancer Heterogeneity, Plasticity and Therapy Resistance, France.ORCID https://orcid.org/0000-0003-0140-6371
Mélanie DhayerUniversity of Lille, CNRS, Inserm, CHU Lille, Institut de Recherche Contre le Cancer de Lille, UMR9020 - UMR-S 1277 - Canther Cancer Heterogeneity, Plasticity and Therapy Resistance, France.
Romain DuboisInstitut de Pahtologie, Centre de Biologie-Pathologie, CHU Lille, France.
Frank LafontUniversity of Lille, CNRS, Inserm, CHU Lille, Institut Pasteur de Lille, U1019 - UMR 9017 - CIIL - Center for Infection and Immunity of Lille, France.
Sebastien JanelUniversity of Lille, CNRS, Inserm, CHU Lille, Institut Pasteur de Lille, U1019 - UMR 9017 - CIIL - Center for Infection and Immunity of Lille, France.
Patrice MaboudouService de Biochimie, Centre de Biologie-Pathologie, CHU Lille, France.
Steve LancelUniversity of Lille, Inserm, CHU Lille, Institut Pasteur de Lille, U1167-RID-AGE-Facteurs de Risque et Déterminants Moléculaires des Maladies Liées au Vieillissement, France.
Philippe MarchettiUniversity of Lille, CNRS, Inserm, CHU Lille, Institut de Recherche Contre le Cancer de Lille, UMR9020 - UMR-S 1277 - Canther Cancer Heterogeneity, Plasticity and Therapy Resistance, France.

Funding

Cancéropole Nord Ouest, Appel à projet EmergenceLigue contre le cancer - comité du Nord and Cancéropole Nord Ouest
6 · The paper itself

Abstract

Myeloid sarcoma (MS) is a rare extramedullary manifestation of acute myeloid leukemia (AML). Conventional two-dimensional cultures fail to replicate the constraints of the MS microenvironment. We demonstrated that 5% gelatin methacrylate (GelMA) hydrogels provide a tunable three-dimensional matrix for in vitro modeling of MS. GelMA hydrogels exhibited tissue-like stiffness and oxygen diffusion properties, supporting the formation of viable spheroids from myeloid leukemia cell lines. The 3D context induced G1 cell cycle arrest and apoptotic progression, reflecting dormancy at extramedullary sites. RNA sequencing revealed transcriptional reprogramming in GelMA-embedded spheroids, with an enrichment in ECM remodeling and metabolic pathways. Furthermore, transcriptomic profiles from 3D-cultured cells aligned with patient-derived MS samples, validating the model's relevance to MS. Thus, we present a GelMA-based system which provides a clinically relevant platform for investigating MS biology and screening potential therapeutic strategies.

Indexed as

bioinkbioprintingchloromaextramedullary myeloid leukemiagranulocytic sarcoma

Identifiers

PMID42592945
PMCPMC13470959

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.