Evidence map›Paper›PMID 42599519›Full record

ArticleEuropean child & adolescent psychiatry2026

Adolescents at the crossroads: genetic associations between psychotic symptoms and antisocial traits.

Natalia Tesli, Piotr Jaholkowski, Jaroslav Rokicki, Andreas Jangmo, Laoura Ziaka, Viktoria Birkenaes, Alexey Shadrin, Martin Tesli, Ole A Andreassen, Unn K Haukvik

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Article in European child & adolescent psychiatry, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
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0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Natalia TesliDivision of Mental Health Services, Akershus University Hospital, Lorenskog, Norway. natalia.tesli@ahus.no.ORCID https://orcid.org/0000-0002-9263-3993
Piotr JaholkowskiCentre for Precision Psychiatry, Division of Mental Health and Addiction, University of Oslo and Oslo University Hospital, Oslo, Norway.
Jaroslav RokickiCentre of Research and Education in Forensic Psychiatry, Oslo University Hospital, Oslo, Norway.
Andreas JangmoDepartment of Chronic Diseases, Norwegian Institute of Public Health, Oslo, Norway.
Laoura ZiakaCentre of Research and Education in Forensic Psychiatry, Oslo University Hospital, Oslo, Norway.
Viktoria BirkenaesCentre for Precision Psychiatry, Division of Mental Health and Addiction, University of Oslo and Oslo University Hospital, Oslo, Norway.
Alexey ShadrinCentre for Precision Psychiatry, Division of Mental Health and Addiction, University of Oslo and Oslo University Hospital, Oslo, Norway.
Martin TesliDepartment of Mental Health and Addiction, Institute of Clinical Medicine, University of Oslo, Oslo, Norway.
Ole A AndreassenCentre for Precision Psychiatry, Division of Mental Health and Addiction, University of Oslo and Oslo University Hospital, Oslo, Norway.
Unn K HaukvikCentre of Research and Education in Forensic Psychiatry, Oslo University Hospital, Oslo, Norway.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Antisocial behaviour in youth has serious consequences at individual and societal level. Adolescence is also a critical period for the emergence of prodromal psychosis symptoms. Their co-occurrence may aggravate aggression and lead to severe adverse outcomes. Common genetic risk for psychosis, antisocial behaviour, and substance use has been suggested, but the putatively shared genetic architecture is unknown, and evidence in youth is limited. Here we examined whether common genetic liability indexed by polygenic risk scores (PRSs) relates to these complex traits in the Norwegian Mother, Father and Child Cohort Study (MoBa; n = 18960). Antisocial psychopathology was operationalised as three complementary dimensions: conduct-disorder (CD) traits, oppositional-defiant disorder (ODD) traits, and psychopathy traits. We first quantified phenotypic correlations between psychotic-like experiences (PLEs) and the antisocial dimensions. We then evaluated PRS-phenotype associations (single-PRS and joint multivariable models) using PRSs for antisocial behaviour (PRS

Indexed as

Antisocial behaviourMoBaPolygenic risk scorePsychotic-like experiences

Identifiers

PMID42599519

What Socratic holds

Textmetadata
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.