ReviewBiomedicines2026
The Role of Endothelial-Mesenchymal Transition (EndMT) in Aneurysm Formation.
Review in Biomedicines, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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0 citing papers in PubMed.
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Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Aneurysms represent a life-threatening disease due to their risk of rupture. Recent research points towards a role of endothelial-mesenchymal transition (EndMT) in their pathophysiology. EndMT acts as a cellular program during which endothelial cells undergo phenotypic and functional transformation into cells with mesenchymal properties. EndMT is implicated in embryogenesis, tissue repair, remodeling, angiogenesis and fibrosis. The studies performed in the last decade have gained new insights in the cell signaling pathways during aneurysm development in the context of EndMT and suggested new drug targets to prevent aneurysmal growth. This review summarizes the current knowledge on EndMT in various forms of aneurysm, such as abdominal and thoracic aortic aneurysms, intracranial aneurysms and coronary artery aneurysm due to Kawasaki disease.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.