Evidence map›Paper›PMID 42766630›Full record

ReviewPLoS pathogens2026

Exploring glycocalyx components powering the contact between Entamoeba histolytica and bacteria during non-opsonic phagocytosis.

Elisabeth Labruyère, Thomas Treport, Jean-Christophe Olivo-Marin, Nancy Guillen

Abstract readReview
In one paragraph

Review in PLoS pathogens, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Elisabeth LabruyèreInstitut Pasteur, Université Paris Cité, Biological Image Analysis Unit, Centre National de la Recherche Scientifique UMR3691, Paris, France.
Thomas TreportInstitut Pasteur, Université Paris Cité, Biological Image Analysis Unit, Centre National de la Recherche Scientifique UMR3691, Paris, France.
Jean-Christophe Olivo-MarinInstitut Pasteur, Université Paris Cité, Biological Image Analysis Unit, Centre National de la Recherche Scientifique UMR3691, Paris, France.
Nancy GuillenInstitut Pasteur, Université Paris Cité, Biological Image Analysis Unit, Centre National de la Recherche Scientifique UMR3691, Paris, France.ORCID https://orcid.org/0000-0001-6551-9012

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Phagocytosis is a cornerstone of the immune defence, allowing cells to eliminate pathogens through both opsonic and non-opsonic pathways. In protozoa such as Entamoeba histolytica, non-opsonic phagocytosis is not only central to survival but also to its ability to cause disease, with its life cycle closely intertwined with the intestinal microbiota, which supplies bacteria and their metabolic byproducts to support trophozoite growth. Yet, despite its significance, the molecular basis of how E. histolytica recognises and engulfs bacteria remains largely unresolved. This review examines the cell-surface molecules that may mediate bacteria-amoeba interactions and highlights new strategies to uncover their roles, offering fresh insight into the dynamics of non-opsonic phagocytosis.

Indexed as

BacteriaEntamoeba histolyticaEntamoebiasisGlycocalyxPhagocytosisAnimalsHumans

Identifiers

PMID42766630
PMCPMC13592706

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.