ReviewNature reviews. Nephrology2026
Primary cilia: master conductors of cellular communication in development and disease.
Review in Nature reviews. Nephrology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
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Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Primary cilia are microtubule-based organelles that function as cellular antennae, integrating diverse signalling pathways essential for tissue development and function. Dysregulation of ciliary signalling is linked to a broad spectrum of disorders known as ciliopathies, which affect most tissues and organs in the body. Primary cilia are dynamic hubs in which selective trafficking and compartmentalization of receptors, effectors and second messengers shape the strength, duration and specificity of cellular responses. Intraflagellar transport, ciliary gating and cargo adaptors establish and dynamically remodel ciliary composition, enabling signalling through Hedgehog, G-protein-coupled receptors, transient receptor potential proteins, receptor tyrosine kinases and the TGFβ superfamily to coordinate developmental patterning, cell proliferation and differentiation, metabolism and tissue homeostasis. Disruption of ciliary architecture, trafficking or signalling can therefore produce diverse and tissue-specific disease phenotypes, including polycystic kidney disease, retinal degeneration, skeletal abnormalities, cardiovascular diseases, neurodevelopmental disorders and obesity. Emerging evidence indicates that cell-type-specific and temporally dynamic ciliary signatures determine signalling competence and disease susceptibility. Defining these signatures and the mechanisms that regulate their plasticity could lead to new therapeutic opportunities for ciliopathies and other cilia-associated diseases.
Identifiers
42768163What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.