ArticleMethods in molecular biology (Clifton, N.J.)2027
Split-GFP Reporter-Based Detection of Chlamydia trachomatis Inclusion Damage.
Article in Methods in molecular biology (Clifton, N.J.), 2027. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Chlamydia trachomatis, an obligate intracellular pathogen responsible for trachoma and widespread urogenital infections, replicates exclusively within the confines of a membrane-bound vacuole known as the inclusion. This compartment shields the bacterium from host cellular immune detection; thus, clarifying how inclusion integrity is maintained and protected from vacuole-destabilizing host attacks may identify new therapeutic strategies. To enable such insights, we recently developed split-GFP-based microscopic reporters that can detect early forms of membrane damage and track individual bacteria released from compromised inclusions. These reporters rely on infecting GFP1-10-expressing cells with C. trachomatis strains engineered to express either a GFP11-tagged outer membrane protein, exposing GFP11 on the bacterial surface, or a GFP11-tagged inclusion membrane protein, exposing GFP11 to the inclusion lumen. Damage to the inclusion allows GFP1-10 to access the bacteria and inner surface of the inclusion membrane, leading to GFP reconstitution and fluorescence at either cytosol-exposed bacteria or damaged membrane sites. Here, we provide detailed protocols for applying these reporter systems, including the generation of the required bacterial strains, development of a GFP1-10-expressing cell line, and execution of a representative infection experiment.
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