ArticleMetabolites2026
Effects of Traffic-Related Diesel Exhaust Exposure and Moderate Exercise on Obesity, Metabolic Dysfunction, and Inflammatory Responses in Rats.
Article in Metabolites, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
3 authors.
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Abstract
objectiveTraffic-related air pollution is recognised as a significant environmental risk factor associated with obesity and metabolic dysfunction. Although the positive effects of regular exercise on metabolic health are well established, its protective role against traffic-related air-pollution-induced metabolic dysfunction has not been fully elucidated. This study was conducted to investigate the effects of diesel exhaust exposure and moderate exercise on the development of obesity, metabolic changes, adipokine profile, and inflammatory response in rats. MATERIALS AND
methodsIn this randomised controlled experimental study, 48 female Wistar Albino rats were randomised into six experimental groups. The animals were exposed to diesel exhaust simulating traffic-related air pollution (average 300 μg PM2.5/m
resultsDiesel exhaust exposure caused a significant increase in final body weight, BMI, Lee index and VAI values (
conclusionsTraffic-related diesel exhaust exposure led to metabolic disorders in rats, characterised by visceral adiposity, dyslipidaemia, impaired glucose homeostasis, adipokine imbalance and chronic low-grade inflammation. Regular moderate-intensity exercise significantly reduced these adverse effects but could not eliminate them entirely. The findings suggest that traffic-related air pollution may contribute to the development of obesity via inflammation-mediated metabolic mechanisms, and that regular physical activity may serve as an important protective strategy in limiting these effects.
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