ArticleJournal of molecular and cellular cardiology plus2026
Systemic inflammation increases mast cell numbers in coronary arteries in patients who died from myocardial infarction.
Article in Journal of molecular and cellular cardiology plus, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Systemic inflammation caused by infection peaks the risk of acute myocardial infarction (AMI), presumably by affecting atherosclerotic plaque (AP) stability. In this study, APs were examined in AMI patients with acute infection (AI). Four groups of AMI patients with or without AI were compared to determine the effects of infection on both early (<6 h) and late (6 h-14 days) AMI: Early AMI, early AMI + AI, late AMI and late AMI + AI. Changes in the AP were determined by measuring stenosis (Elastica von Gieson), plaque stability (thickness of fibrotic cap and leukocytes presence) and intraplaque haemorrhage (GLUT-1 and Perls). Inflammation was determined by measuring leukocytes, macrophages, mast cells and neutrophilic granulocytes in the different regions of the coronary arteries: intima, media and adventitia. AI did not affect coronary stenosis, plaque stability or intraplaque haemorrhage. Plaque instability increased in late AMI + AI compared to early AMI + AI (
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