ArticleThe Journal of clinical investigation1994

Mutations of CpG dinucleotides located in the triiodothyronine (T3)-binding domain of the thyroid hormone receptor (TR) beta gene that appears to be devoid of natural mutations may not be detected because they are unlikely to produce the clinical phenotype of resistance to thyroid hormone.

Y Hayashi et al.PubMed ↗Full text ↗Publisher ↗

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