Evidence mapPaperPMID 8675665Full record

ArticleThe Journal of clinical investigation1995

A genetic model for absent chylomicron formation: mice producing apolipoprotein B in the liver, but not in the intestine.

S G Young, C M Cham, R E Pitas, B J Burri, A Connolly, L Flynn, A S Pappu, J S Wong, R L Hamilton, R V Farese

Open access · bronzeAbstract read
In one paragraph

Article in The Journal of clinical investigation, 1995. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 27 papers.

0numbers the graph read from it
0cells of the map it votes in
27citing papers in PubMed
1.5field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

27 citing papers in PubMed, 125 citations in OpenAlex.

  1. Trial
  2. Oral Glucose Mobilizes Triglyceride Stores From the Human Intestine.Cellular and molecular gastroenterology and hepatology · 2019
    Trial
  3. Article
  4. Article
  5. Article
  6. Article
  7. Review
  8. Article
  9. Review
  10. Review
  11. Review
  12. Article
  13. Review
  14. An apolipoprotein B100 mimotope prevents obesity in mice.Clinical science (London, England : 1979) · 2016
    Article
  15. Review
  16. Article
  17. Review
  18. Article
  19. Article
  20. Intestinal lymphatic transport for drug delivery.Advanced drug delivery reviews · 2011
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

S G YoungGladstone Institute of Cardiovascular Disease, San Francisco, California 94141-9100, USA.
C M Cham
R E Pitas
B J Burri
A Connolly
L Flynn
A S Pappu
J S Wong
R L Hamilton
R V Farese
University of California, San Francisco · USGladstone Institutes · US

Funding

TRANSGENIC ANIMAL MODEL OF TYPE III HYPERLIPOPROTEINEMIAP01HL047660 · J. DAVID GLADSTONE INSTITUTES · 1992 to 2001
$2.9M
NHLBI NIH HHS P01HL-47660
6 · The paper itself

Abstract

The formation of chylomicrons by the intestine is important for the absorption of dietary fats and fat-soluble vitamins (e.g., retinol, alpha-tocopherol). Apo B plays an essential structural role in the formation of chylomicrons in the intestine as well as the VLDL in the liver. We have developed genetically modified mice that express apo B in the liver but not in the intestine. By electron microscopy, the enterocytes of these mice lacked nascent chylomicrons in the endoplasmic reticulum and Golgi apparatus. Because these mice could not form chylomicrons, the intestinal villus enterocytes were massively engorged with fat, which was contained in cytosolic lipid droplets. These mice absorbed D-xylose normally, but there was virtually no absorption of retinol palmitate or cholesterol. The levels of alpha-tocopherol in the plasma were extremely low. Of note, the absence of chylomicron synthesis in the intestine did not appear to have a significant effect on the plasma levels of the apo B-containing lipoproteins produced by the liver. The mice lacking intestinal apo B expression represent the first genetic model of defective absorption of fats and fat-soluble vitamins and provide a useful animal model for studying nutrition and lipoprotein metabolism.

Indexed as

AnimalsApolipoproteins BChylomicronsCrosses, GeneticDiterpenesEndoplasmic ReticulumFemaleGenotypeGolgi ApparatusHeterozygoteHumansIntestinal AbsorptionIntestinal MucosaIntestinesLiverMaleApolipoproteins BChylomicronsDiterpenesretinol palmitateRetinyl EstersVitamin AVitamin E

Identifiers

PMID8675665
PMCPMC186005
OpenAlexW1996152427

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.