Evidence mapPaperPMID 9022076Full record

Trial reportThe Journal of clinical investigation1997

The vascular effects of L-Arginine in humans. The role of endogenous insulin.

D Giugliano, R Marfella, G Verrazzo, R Acampora, L Coppola, D Cozzolino, F D'Onofrio

Abstract readClinical Trial
In one paragraph

Trial report in The Journal of clinical investigation, 1997. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Trial
  3. Trial
  4. Trial
  5. Trial
  6. Trial
  7. Article
  8. Article
  9. Article
  10. Review
  11. Article
  12. Article
  13. Impact of postprandial glycaemia on health and prevention of disease.Obesity reviews : an official journal of the International Association for the Study of Obesity · 2012
    Review
  14. Article
  15. Article
  16. Article
  17. Article
  18. Article
  19. Translational control of inducible nitric oxide synthase expression by arginine can explain the arginine paradox.Proceedings of the National Academy of Sciences of the United States of America · 2003
    Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

D GiuglianoDepartment of Geriatrics and Metabolic Diseases, Second University of Naples, Italy.
R Marfella
G Verrazzo
R Acampora
L Coppola
D Cozzolino
F D'Onofrio

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

This study aimed at evaluating whether increased availability of the natural precursor of nitric oxide, L-arginine, could influence systemic hemodynamic and rheologic parameters in humans and whether the effects of L-arginine are mediated by endogenous insulin. 10 healthy young subjects participated in the following studies: study I, infusion of L-arginine (1 g/min for 30 min); study II, infusion of L-arginine plus octreotide (25 microg as i.v. bolus + 0.5 microg/min) to block endogenous insulin and glucagon secretion, plus replacement of basal insulin and glucagon; study III, infusion of L-arginine plus octreotide plus basal glucagon plus an insulin infusion designed to mimic the insulin response of study I. L-Arginine infusion significantly reduced systolic (11+/-3, mean+/-SE) and diastolic (8+/-2 mmHg, P < 0.001) blood pressure, platelet aggregation (20+/-4%), and blood viscosity (1.6+/-0.2 centipois, P < 0.01), and increased leg blood flow (97+/-16 ml/min), heart rate, and plasma catecholamine levels (P < 0.01). In study II, plasma insulin levels remained suppressed at baseline; in this condition, the vascular responses to L-arginine were significantly reduced, except for plasma catecholamines which did not change significantly. In study III, the plasma insulin response to L-arginine was reestablished; this was associated with hemodynamic and rheologic changes following L-arginine not significantly different from those recorded in study I. These findings show that systemic infusion of L-arginine in healthy subjects induces vasodilation and inhibits platelet aggregation and blood viscosity. These effects are mediated, in part, by endogenous released insulin.

Indexed as

AdultArginineBlood PressureBlood ViscosityCatecholaminesFemaleGastrointestinal AgentsGlucagonHeart RateHemodynamicsHormonesHumansHypoglycemic AgentsInsulinLegMaleArginineCatecholaminesGastrointestinal AgentsGlucagonHormonesHypoglycemic AgentsInsulinNitric OxideOctreotide

Identifiers

PMID9022076
PMCPMC507816

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.