Evidence map›Paper›PMID 9042915›Full record

ArticleAmerican journal of human genetics1997

Partial correction of a severe molecular defect in hemophilia A, because of errors during expression of the factor VIII gene.

M Young, H Inaba, L W Hoyer, M Higuchi, H H Kazazian, S E Antonarakis

Abstract read
In one paragraph

Article in American journal of human genetics, 1997. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed
1.1field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 94 citations in OpenAlex.

  1. Spectrum of Molecular Defects in 216 Chinese Families With Hemophilia A: Identification of Noninversion Mutation Hot Spots and 42 Novel Mutations.Clinical and applied thrombosis/hemostasis : official journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis · 2018
    Trial
  2. Article
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  4. Mutation analysis in the F8 gene in 485 families with haemophilia A and prenatal diagnosis in China.Haemophilia : the official journal of the World Federation of Hemophilia · 2021
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  19. Unexpected frameshifts from gene to expressed protein in a phage-displayed peptide library.Proceedings of the National Academy of Sciences of the United States of America · 1998
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

M YoungDepartment of Genetics and Microbiology, University of Geneva, Switzerland.
H Inaba
L W Hoyer
M Higuchi
H H Kazazian
S E Antonarakis
University of Geneva · CH

Funding

STUDIES OF ANTIBODIES TO ANTIHEMOHILIC FACTORR01HL036099 · NHLBI · AMERICAN NATIONAL RED CROSS · PI HOYER, LEON W. · 1985 to 1998
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MOLECULAR GENETICS OF HEMOPHILIA AR01HL038165 · NHLBI · UNIVERSITY OF PENNSYLVANIA · PI KAZAZIAN, HAIG H. · 1987 to 1999
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NHLBI NIH HHS HL36099NHLBI NIH HHS HL38165
6 · The paper itself

Abstract

Although the molecular defect in patients in a Japanese family with mild to moderately severe hemophilia A was a deletion of a single nucleotide T within an A8TA2 sequence of exon 14 of the factor VIII gene, the severity of the clinical phenotype did not correspond to that expected of a frameshift mutation. A small amount of functional factor VIII protein was detected in the patient's plasma. Analysis of DNA and RNA molecules from normal and affected individuals and in vitro transcription/translation suggested a partial correction of the molecular defect, because of the following: (i) DNA replication/RNA transcription errors resulting in restoration of the reading frame and/or (ii) "ribosomal frameshifting" resulting in the production of normal factor VIII polypeptide and, thus, in a milder than expected hemophilia A. All of these mechanisms probably were promoted by the longer run of adenines, A10 instead of A8TA2, after the delT. Errors in the complex steps of gene expression therefore may partially correct a severe frameshift defect and ameliorate an expected severe phenotype.

Indexed as

Frameshift MutationDNA ReplicationExonsFactor VIIIFemaleFrameshifting, RibosomalGene ExpressionHemophilia AHumansMalePedigreeProtein BiosynthesisReading FramesTemplates, GeneticTranscription, GeneticFactor VIII

Identifiers

PMID9042915
PMCPMC1712533
OpenAlexW2098857164

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.