Evidence map›Paper›PMID 9528793›Full record

ArticleMolecular and cellular biology1998

A role for CREB binding protein and p300 transcriptional coactivators in Ets-1 transactivation functions.

C Yang, L H Shapiro, M Rivera, A Kumar, P K Brindle

Open access · greenAbstract read
In one paragraph

Article in Molecular and cellular biology, 1998. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 79 papers.

0numbers the graph read from it
0cells of the map it votes in
79citing papers in PubMed
7.6field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

79 citing papers in PubMed, 219 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. ETS1 Function in Leukemia and Lymphoma.Advances in experimental medicine and biology · 2024
    Review
  6. Article
  7. Article
  8. Review
  9. Review
  10. Article
  11. Article
  12. Article
  13. Article
  14. ETS-targeted therapy: can it substitute for MEK inhibitors?Clinical and translational medicine · 2017
    Review
  15. Activation of MouseJournal of immunology (Baltimore, Md. : 1950) · 2017
    Article
  16. Article
  17. Article
  18. Article
  19. Article
  20. Article

19 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 3 countries.

C YangDepartment of Biochemistry, St. Jude Children's Research Hospital, Memphis, Tennessee 38105, USA.
L H Shapiro
M Rivera
A Kumar
P K Brindle
Laboratoire de Biochimie · FRSt. Jude Children's Research Hospital · US

Funding

Viral Vector Technology (VVTSR)P30CA021765 · NCI · ST. JUDE CHILDREN'S RESEARCH HOSPITAL · PI Markos Leggas · 1985 to 2026
$166.9M
TRANSCRIPTIONAL COACTIVATORS IN HEMATOPOIESISR01CA076385 · NCI · ST. JUDE CHILDREN'S RESEARCH HOSPITAL · PI BRINDLE, PAUL K · 1998 to 2005
$1.4M
MYB, ETS, AML-1 AND AML-ETO IN ACUTE MYELOID LEUKEMIAR29CA070909 · NCI · ST. JUDE CHILDREN'S RESEARCH HOSPITAL · PI SHAPIRO, LINDA H · 1996 to 2000
$107k
NCI NIH HHS CA70909NCI NIH HHS P30 CA021765NCI NIH HHS P30 CA21765NCI NIH HHS R01 CA076385NCI NIH HHS R01 CA76385
6 · The paper itself

Abstract

The Ets-1 transcription factor plays a critical role in cell growth and development, but the means by which it activates transcription are still unclear (J. C. Bories, D. M. Willerford, D. Grevin, L. Davidson, A. Camus, P. Martin, D. Stehelin, F. W. Alt, and J. C. Borles, Nature 377:635-638, 1995; N. Muthusamy, K. Barton, and J. M. Leiden, Nature 377:639-642, 1995). Here we show that Ets-1 binds the transcriptional coactivators CREB binding protein (CBP) and the related p300 protein (together referred to as CBP/p300) and that this interaction is required for specific Ets-1 transactivation functions. The Ets-1- and c-Myb-dependent aminopeptidase N (CD13/APN) promoter and an Ets-1-dependent artificial promoter were repressed by adenovirus E1A, a CBP/p300-specific inhibitor. Furthermore, Ets-1 activity was potentiated by CBP and p300 overexpression. The transactivation function of Ets-1 correlated with its ability to bind an N-terminal cysteine- and histidine-rich region spanning CBP residues 313 to 452. Ets-1 also bound a second cysteine- and histidine-rich region of CBP, between residues 1449 and 1892. Both Ets-1 and CBP/p300 formed a stable immunoprecipitable nuclear complex, independent of DNA binding. This Ets-1-CBP/p300 immunocomplex possessed histone acetyltransferase activity, consistent with previous findings that CBP/p300 is associated with such enzyme activity. Our results indicate that CBP/p300 may mediate antagonistic and synergistic interactions between Ets-1 and other transcription factors that use CBP/p300 as a coactivator, including c-Myb and AP-1.

Indexed as

Saccharomyces cerevisiae ProteinsTrans-ActivatorsTranscriptional ActivationAcetyltransferasesCD13 AntigensCell LineCREB-Binding ProteinDNAHistone AcetyltransferasesHumansJurkat CellsNuclear ProteinsPromoter Regions, GeneticProtein BindingProto-Oncogene Protein c-ets-1Proto-Oncogene ProteinsAcetyltransferasesCD13 AntigensCREB-Binding ProteinCREBBP protein, humanDNAETS1 protein, humanHistone AcetyltransferasesNuclear ProteinsProto-Oncogene Protein c-ets-1Proto-Oncogene ProteinsProto-Oncogene Proteins c-etsSaccharomyces cerevisiae ProteinsTrans-ActivatorsTranscription Factors

Identifiers

PMID9528793
PMCPMC121466
OpenAlexW2156860267

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.