Evidence mapPaperPMID 12949632Full record

ReviewMethods and findings in experimental and clinical pharmacology

Clinical pharmacokinetics of statins.

M J García, R F Reinoso, A Sánchez Navarro, J R Prous

Registry-linked trialAbstract readComparative StudyReview
PubMed
In one paragraph

Review in Methods and findings in experimental and clinical pharmacology. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It reports registered trial NCT01634906. Cited by 37 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
37citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT01634906 nacompleted

Erythrocyte-bound Apolipoprotein B After Withdrawal of Statin Therapy

Ran2012Enrolled55Registered outcomes3Posted comparisons0ConditionsAtherosclerosis, HyperlipidemiaArmsTemporary discontinuation of statin therapy
Open the trial in the graph
3 · Its place in the literature

Who cites it

37 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Trial
  3. Trial
  4. Review
  5. Article
  6. Article
  7. HMG-CoA Reductase Inhibitors for Traumatic Brain Injury.Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics · 2023
    Review
  8. Article
  9. Article
  10. Simvastatin-loaded liposome nanoparticles treatment for uterine leiomyoma in a patient-derived xenograft mouse model: a pilot study.Journal of obstetrics and gynaecology : the journal of the Institute of Obstetrics and Gynaecology · 2022
    Article
  11. Article
  12. Article
  13. Review
  14. Pharmaceutical Excipients and Drug Metabolism: A Mini-Review.International journal of molecular sciences · 2020
    Review
  15. Review
  16. Article
  17. Article
  18. Article
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

M J GarcíaDepartment of Pharmacy and Pharmaceutical Technology, Salamanca, Spain.
R F Reinoso
A Sánchez Navarro
J R Prous

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

This article reviews the pharmacokinetic properties of HMG-CoA reductase inhibitors (or statins), as reported in humans. Most data presented here refer to commercially available statins (atorvastatin, fluvastatin, lovastatin and simvastatin), although statins that have recently been withdrawn (cerivastatin) or are currently under development (glenvastatin, pitavastatin and rosuvastatin) will also be considered. All statins with the exception of pitavastatin show very low systemic bioavailability due to an extensive first pass effect at the intestinal and/or hepatic level. Such a characteristic can be advantageous, since the liver is the target organ for statins. Unlike most statins, lovastatin and simvastatin are administered as inactive lactone prodrugs. Statins differ mainly in the degree of metabolism and the number of active and inactive metabolites. All statins but pravastatin show highly active metabolites, the pharmacological activity depending on the kinetic profile of both parent compound and active metabolites. Pravastatin has the lowest protein binding (50% vs. > 90%) and is eliminated by both metabolism and renal excretion. Atorvastatin shows the longest terminal half-life (11-14 h vs. 1-3 h). Pharmacokinetic interactions with statins are very likely to occur, particularly for those statins that are CYP3A4 substrates. However, although of extreme interest in clinical practice, this subject was extensively reviewed in a previous article and therefore is not discussed here.

Indexed as

Administration, OralAge FactorsAnticholesteremic AgentsBiological AvailabilityClinical Trials as TopicFemaleHumansHydroxymethylglutaryl-CoA Reductase InhibitorsMaleSex FactorsAnticholesteremic AgentsHydroxymethylglutaryl-CoA Reductase Inhibitors

Identifiers

PMID12949632

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.