Evidence map›Paper›PMID 8997227›Full record

ArticleThe American journal of physiology1996

Effect of perfusion rate on the time course of insulin-mediated skeletal muscle glucose uptake.

A D Baron, G Brechtel-Hook, A Johnson, J Cronin, R Leaming, H O Steinberg

2 registry-linked trialsAbstract read
PubMed Publisher
In one paragraph

Article in The American journal of physiology, 1996. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to 2 registered trials, which are not on this map. Cited by 36 papers.

0numbers the graph read from it
0cells of the map it votes in
36citing papers in PubMed
7.5field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02633488 nacompletedstarted 2014, after this paper: background citation

Effect of Metformin on Insulin Sensitivity and Pan-Arterial Vascular Function in Adults With Metabolic Syndrome

Ran2014Enrolled19Registered outcomes1Posted comparisons1ConditionsInsulin Sensitivity, Metabolic SyndromeArmsMetformin, Placebos
Open the trial in the graph
NCT04133922 early_phase1withdrawnstarted 2019, after this paper: background citation

Effect of GLP-1 on Microvascular Insulin Responses in Type 1 Diabetes

Ran2019Enrolled0Registered outcomes5Posted comparisons0ConditionsInsulin Sensitivity/Resistance, Type 1 DiabetesArmsDextrose 20 % in Water, GLP-1, Insulin
Open the trial in the graph
3 · Its place in the literature

Who cites it

36 citing papers in PubMed, 95 citations in OpenAlex.

  1. Trial
  2. Trial
  3. Article
  4. Article
  5. An oral glucose tolerance test does not affect cerebral blood flow: role of NOS.American journal of physiology. Regulatory, integrative and comparative physiology · 2023
    Article
  6. Sympathetic transduction to blood pressure during euglycemic-hyperinsulinemia in young healthy adults: role of burst amplitude.American journal of physiology. Regulatory, integrative and comparative physiology · 2023
    Article
  7. New insights into mechanisms of endothelial insulin resistance in type 2 diabetes.American journal of physiology. Heart and circulatory physiology · 2022
    Review
  8. Article
  9. ADAM17 cleaves the insulin receptor ectodomain on endothelial cells and causes vascular insulin resistance.American journal of physiology. Heart and circulatory physiology · 2022
    Article
  10. Article
  11. Article
  12. Insulin stimulation reduces aortic wave reflection in adults with metabolic syndrome.American journal of physiology. Heart and circulatory physiology · 2021
    Article
  13. Hyperinsulinemia blunts sympathetic vasoconstriction: a possible role of β-adrenergic activation.American journal of physiology. Regulatory, integrative and comparative physiology · 2021
    Article
  14. Review
  15. Article
  16. Article
  17. Review
  18. Review
  19. Article
  20. The cell biology of systemic insulin function.The Journal of cell biology · 2018
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

A D BaronDepartment of Medicine, Indiana University Medical Center, Indianapolis, USA.
G Brechtel-Hook
A Johnson
J Cronin
R Leaming
H O Steinberg
Indiana University – Purdue University Indianapolis · US

Funding

THE CLINTON COUNTY OUTREACH PROJECT (CCOP)P60DK020542 · NIDDK · INDIANA UNIV-PURDUE UNIV AT INDIANAPOLIS · PI CLARK, CHARLES MALCOLM · 1986 to 2003
$5.1M
ROLE OF HEMODYNAMICS IN IN VIVO INSULIN RESISTANCER37DK042469 · NIDDK · INDIANA UNIV-PURDUE UNIV AT INDIANAPOLIS · PI MATHER, KIEREN J · 1999 to 2003
$1.1M
ROLE OF HEMODYNAMICS IN IN VIVO INSULIN RESISTANCER01DK042469 · NIDDK · INDIANA UNIV-PURDUE UNIV AT INDIANAPOLIS · PI BARON, ALAIN D · 1995 to 1998
–
ROLE OF HEMODYNAMICS IN IN VIVO INSULIN RESISTANCER29DK042469 · NIDDK · INDIANA UNIV-PURDUE UNIV AT INDIANAPOLIS · PI BARON, ALAIN D · 1990 to 1994
–
NCRR NIH HHS MO1-RR750NIDDK NIH HHS DK-20542NIDDK NIH HHS DK-42469
6 · The paper itself

Abstract

To better define the time course of skeletal muscle glucose uptake and its modulation by changes in perfusion, we performed systemic euglycemic-hyperinsulinemic clamps (40 mU.m-2.min-1) for a 90-min period in a group of lean, insulin-sensitive subjects (n = 9) on two occasions (approximately 4 wk apart) with insulin-mediated vasodilation intact or inhibited. Insulin-mediated vasodilation was inhibited by an intrafemoral artery infusion of NG-monomethyl-L-arginine (L-NMMA), a specific inhibitor of nitric oxide synthase. During the study, leg blood flow (LBF) and arteriovenous glucose difference (AVG delta) were measured every 10 min; leg glucose uptake (LGU) was calculated as LGU = LBF x AVG delta. The systemic insulin infusion caused a time-dependent increase in LBF from 0.194 +/- 0.024 to 0.349 +/- 0.046 l/min (P < 0.01). The intrafemoral artery infusion of L-NMMA completely inhibited this increase in LBF. AVG delta, LGU, and whole body glucose disposal rates increased in a time-dependent manner in both studies. The maximum AVG delta was lower with insulin-mediated vasodilation intact than when inhibited (25.9 +/- 2.5 vs. 35.0 +/- 1.6 mg/dl, P < 0.001). The time to achieve half-maximal (T1/2) AVG delta was somewhat longer with insulin-mediated vasodilation intact compared with inhibited (35.6 +/- 4.1 vs. 29.7 +/- 1.6 min, P < 0.01). Maximal LGU was 93.9 +/- 26.8 and 57.2 +/- 11.6 mg/min (P < 0.005), and the T1/2 LGU was 50.2 +/- 16.0 and 36.3 +/- 8.8 min (P = 0.1) during intact and inhibited insulin-mediated vasodilation, respectively. Thus insulin-mediated vasodilation has a modest effect in slowing the time course at which insulin stimulates glucose uptake but has a marked effect in augmenting the maximal rate of insulin-stimulated glucose uptake in skeletal muscle. Impaired insulin-mediated vasodilation, as observed in patients with essential hypertension, may explain, at least in part, the insulin resistance observed in these patients.

Indexed as

AdultEnzyme InhibitorsFemaleGlucoseHumansHypoglycemic AgentsInsulinMaleMuscle, Skeletalomega-N-MethylargininePerfusionTime FactorsEnzyme InhibitorsGlucoseHypoglycemic AgentsInsulinomega-N-Methylarginine

Identifiers

PMID8997227
OpenAlexW2405570732

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.